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Year : 2022  |  Volume : 20  |  Issue : 3  |  Page : 187-190

Dimethoate self-poisoning–Induced severe hypotension

Department of Neurointensive Care and Trauma Unit, Ruby Hall Clinic, Pune, Maharashtra, India

Correspondence Address:
Dr. Anand M Tiwari
Department of Neurointensive Care and Trauma Unit, Ruby Hall Clinic, Pune, Maharashtra
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Source of Support: None, Conflict of Interest: None

DOI: 10.4103/cmi.cmi_12_22

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Intentional ingestion of organophosphorus compound (OP) poisoning continues to be a frequent reason for critical care admission in India. For the past 6 decades, the OP compounds used in India for the poisoning had effects on cholinergic and neurogenic fibers producing the respiratory and neurologic presentation. Recently, a less frequent OP poison has appeared which acts on cardiac sympathetic fibers with bradycardia and hypotension as presenting features. The shock which was nonresponsive to dopamine/dobutamine was identified as a major cause of death in patients with dimethoate poisoning in a large prospective cohort study. Severe dimethoate poisoning in contrast to other OP compounds has a high case fatality rate. There have been case reports from outside India. We report the first two cases with successful treatment of this toxicity in India.

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